Govind Shukla, Specializes in Pharmacology, Toxicology, Nutraceuticals & Herbal Drugs has published More than 100 research papers in National & International Journals. He is also a reviewer of International Journal of Pharmacology & pharmacotherapeutics, Chief editor of IJPNR Journal & Freelance Medical Writer for Different publication Groups including Lambert Academic Publishing Saarbrucken, Germany.

FERA-Q tablets Helps to support the treatment of Cancer induced Anaemia in patients undergoing chemotherapy and radiotherapy

Cancer-induced anemia is most common in patients with cancer and in patients undergoing chemotherapy and radiation therapy. The severity of anemia depends on the extent of disease and the density of treatment. Repeated cycles of chemotherapy may impair erythropoiesis synthesis that results in decrease functional capacity and quality of life. In cancer patients, the incidence of anemia may rise 40%.According to European cancer anemia survey (ECAS) 53% of patients did not receive any treatment for their anemia.Anemia has also been identified as an adverse prognostic factor for these patients.thus Anemia exerts a negative influence on the quality of life of cancer patients as it may contribute to cancer-induced fatigue.

Mechanism of Action of fera-Q tablets.

Zinc in fera-Q tablets : Zinc in fera-Q tablets shows regulatory role in to gene expression, cell signaling, and nerve impulse transmission, as well as normal apoptosis.. Zinc plays an important role in the structure of proteins and cell membranes. Loss of zinc from biological membranes increases their susceptibility to oxidative damage and impairs their function.

Zinc’s role in supporting immune function includes regulating T lymphocytes, natural killer cells, CD4 cells, and interleukin-2. The mechanism of zinc inhibition of prostatic cell growth involved the effect of zinc on cell cycle progression (cell arrested G2/M) and apoptosis.

Vitamin B6 in fera-Q tablets : Vitamin B6 is a water-soluble vitamin that participates in more than 100 coenzyme reactions involved in the metabolism of protein, carbohydrates, and lipids. Plasma pyridoxal 5’-phosphate (PLP) is the active form of vitamin B6. Vitamin B6 may influence colorectal carcinogenesis through its role in DNA synthesis and methylation. Vitamin B6 has been shown to inhibit angiogenesis, suppress nitric oxide, and reduce oxidative stress, all of which are associated with preventing carcinogenesis.

DNA methylation plays an essential role during normal development, however, it is also considered to be a critical process in the development of several forms of cancer. Dietary methyl (folate, choline, and methionine) deficiency in combination causes decreased tissue S-adeno-sylmethionine, global DNA hypomethylation, hepatic steatosis, cirrhosis, and ultimately hepatic tumorigenesis in rodents in the absence of carcinogen treatment

Methyl folate in fera-Q tablets : Methyl folate helps to regulate gene activity by controlling gene methylation there by it helps in cancer prevention.Methylcobalamin and 5’-deoxyadenosylcobalamin, as sources for vitamin B12 in fera-Q tablets  is involved in synthesis of phospholipids, neurotransmitters, DNA, and the metabolism of fatty acids and amino acids in cells

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